The second critical aspect of HMN147 work is its effect on neurotrophins. In vitro studies on cortical neurons suggest that exposure to HMN147 upregulates the expression of and its high-affinity receptor, TrkB .
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: The mutation causes high penetrance of defects, meaning a large percentage of individuals with this genotype will show physical abnormalities in their neural wiring. Broader Scientific Significance The second critical aspect of HMN147 work is
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The most robust hypothesis regarding HMN147 work involves the . Researchers theorize that HMN147 acts as a positive allosteric modulator (PAM) of nicotinic acetylcholine receptors (nAChRs), specifically the α7 subtype.